Mangiferin Attenuates Aflatoxin B1-Induced Ferroptosis by Activating of Nrf2-Signaling and Mitochondrial Biogenesis in Human Ovarian Granulosa Cells.
The mechanisms by which MA attenuates AFB1-induced oxidative stress and ferroptosis in granulosa cells through the activation of Nrf2 signaling and mitochondrial biogenesis are examined, highlighting the potential of MA as a nutraceutical candidate for alleviating AFB1-induced granulosa cell toxicity.