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Author

F. Guo

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Open access Aug 2026

Injury-transduced oligodendrocytes modulate neuroinflammation and glial activation in diseased and non-diseased central nervous system

It is shown that OLs respond to demyelinating diseases by increasing the expression and secretion of serine protease inhibitor clade A member 3N (SERPINA3N), and that SerpinOLs represent a common population of injury-transduced OLs that contributes to CNS pathology beyond myelin production.

Yan Wang, Mei-Na Zhu, Joohyun Park et al. · 0 citations
Review Open access Aug 2026

Crosstalk Between Oligodendrocyte Lineage Cells and CNS‐Resident and Peripheral Immune Cells Governs Demyelination and Remyelination in Multiple Sclerosis

Current knowledge of OLC biology in health and disease is synthesized, with emphasis on complex intercellular interactions that determine demyelination, remyelination, and axonal integrity in MS.

Joohyun Park, So Yeong Cheon, F. Guo · 0 citations

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