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The vital role of mitophagy in kidney diseases is examined, the potential of mitophagy-targeted therapies are discussed, and detailed alterations in specific mitophagy-related proteins associated with kidney diseases are described.
This study provides the first multi-omics characterization of MMT in bladder cancer, identifies KLF6 as a previously unrecognized driver of this transition, and demonstrates that KLF6-driven MMT upregulates tumour PD-L1 through intercellular crosstalk.
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