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Aggregation, Propagation and Pathological Implications of α-synuclein Amyloid Fibrils in Parkinson’s Disease
It is shown that α-synuclein fibrils are more structurally heterogenous than previously believed and that they may continue to remodel their architecture over the course of PD, and fibril assembly pathways and resulting fibril structure are highlighted as critical targets for future therapeutic strategies.
Fritjof Havemeister
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