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Author

Fu-Tong Liu

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Aug 2026

Galectin-3 exacerbates influenza a virus pathogenesis via dual mechanisms: Promoting hemagglutinin-dependent entry and driving nlrp3 inflammasome activation.

Gal3 is identified as a potent host factor that orchestrates excessive inflammation by simultaneously facilitating viral entry and driving the assembly of the NLRP3 inflammasome, highlighting the Gal3-NLRP3 axis as a promising therapeutic target to mitigate severe influenza-induced lung injury.

Zih-Syuan Yang, A. Thitithanyanont, Jason C. Huang et al. · 0 citations

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