Aug 2026
IP3R drives cardiomyocyte injury by enhancing MAM-mediated Ca2+ transfer and mitochondrial dysfunction.
Findings suggest that IP3R may exacerbate cardiomyocyte injury by facilitating MAM-mediated Ca2+ transfer and mitochondrial dysfunction, indicating that targeting IP3R could represent a potential therapeutic strategy for HF.
Haihui Yang, Jin-Ming Zhu, Gui-Fang Li et al.
· Molecular and Cellular Endoc... · 0 citations