Author

Il-Young Kim

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Open access Aug 2026

Exogenous lactate ameliorates Aβ-induced energy deficit and neurotoxicity with increased mitochondrial TCA cycle carbon flux in SH-SY5Y cells.

A growing body of evidence has demonstrated the existence of metabolic dysfunction in neurodegenerative diseases, including Alzheimer's disease (AD), suggesting that deprivation of energy substrates impairs cellular dynamics. As the glucose utilization declines in AD patients, the need for alternative energy sources be...

Yewon Chang, Hee-Joo Kim, Yeongmin Kim et al. · American Journal of Physiolo... · 0 citations

Open access Sep 2026

Inotodiol prevents age-related muscle wasting by restoring mitochondrial function through liver X receptor beta signaling

Ino treatment significantly improved muscle mass and function in aged mice and prevented dexamethasone (DEX)-induced muscle atrophy and is identified as a promising therapeutic candidate for preserving mitochondrial function and alleviating age-associated muscle dysfunction.

Chang-Lim You, Ju-Hyeon Bae, Yideul Jeong et al. · Signal Transduction and Targ... · 0 citations

Open access Aug 2026

Meloxicam protects against muscle and metabolic decline in ageing through Cdon restoration and oxidative stress modulation

Meloxicam (Mcam) is identified as a small molecule inducer of Cdon that enhances myogenic differentiation, increases muscle mass and function in young mice and mitigates age-related muscle atrophy, and is identified as a potential therapeutic target for age-associated skeletal muscle degeneration.

Ju-Hyeon Bae, Hyun-Kyung So, Yideul Jeong et al. · Experimental and Molecular M... · 0 citations