Cancer-Associated Fibroblasts-Derived Exosomal METTL3 Drives Breast Cancer Malignancy by Eliciting PABPN1 m6A Modification.
A novel CAFs-to-cancer cell communication axis where exosomal METTL3 promotes BC progression and immune escape via m6A-dependent upregulation of PABPN1 is identified, highlighting this pathway as a promising therapeutic target.