CREG1 mitigates sepsis-induced acute lung injury by mediating macrophage polarization via AMPK/NF-κB signaling.
BACKGROUND Sepsis-induced acute lung injury (ALI) is a severe respiratory disease that usually progresses to acute respiratory distress syndrome (ARDS). Cellular repressor of E1A-stimulated genes 1 (CREG1), a secreted glycoprotein, participates in tissue homeostasis, influences inflammation, and improves cardiovascular...