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Author

Min-Kyeong Ju

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Open access Jul 2026

TLR4-dependent upregulation of IL-22-producing T cells in the bone niche links to the imbalance of bone homeostasis.

Evidence is provided that IL-22 acts as a TLR4-driven mediator to shift bone remodeling toward catabolic bone degeneration and co-culture of pre-osteoblasts and BM cells supported that IL-22 acts as a TLR4-driven mediator to shift bone remodeling toward catabolic bone degeneration.

Ji-Eun Kim, Jinyoung Go, Eun-Jin Lee et al. · 0 citations

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