miR-222-3p inhibits diabetic skin wound healing by targeting APLN
Results suggest that miR-222-3p impairs microvascular endothelial cell function by targeting APLN, thereby contributing to delayed wound healing in diabetic skin.
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Results suggest that miR-222-3p impairs microvascular endothelial cell function by targeting APLN, thereby contributing to delayed wound healing in diabetic skin.
ABSTRACT Aims To investigate the role of RNA N6‐methyladenosine (m6A) methyltransferase Wilms tumor 1‐associated protein (WTAP) in negative pressure wound therapy (NPWT) for diabetic foot ulcers (DFU). Materials and Methods We enrolled 46 DFU patients and 16 non‐diabetic chronic ulcer patients treated with NPWT. Total...
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