Review
Open access
Sep 2026
Gut Dysbiosis, Uremic Toxins, and NLRP3 Signaling in Chronic Kidney Disease: From Mechanistic Evidence to Clinical Biomarker Evaluation
The gut–toxin–inflammasome axis is mechanistically plausible, but evidence for priming is substantially stronger than that for direct activation and evidence for priming is substantially stronger than that for direct activation.
Imola Donath-Miklos, O. Știrbu, R. Popețiu et al.
· Diagnostics · 0 citations