AI Networking Cookbook: Practical recipes for AI-assisted network automation and development
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It is demonstrated that disruption of neurexin–neuroligin signaling drives transcriptional reprogramming that extends beyond synaptic dysfunction, linking molecular alterations to developmental, behavioral, and neuromorphological abnormalities.
This review integrates findings from studies across various model organisms to elucidate how alterations in synaptic function disrupt social interaction, and discusses the use of drugs and gene therapies in the management and treatment of the symptoms observed in social interaction disorders.
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