Open access
Sep 2026
Deletion of the T1D risk gene Ormdl3 in β-cells does not impact disease progression
Findings indicate that ORMDL3 function in β-cells is dispensable in this preclinical model of T1D and underscore the need to investigate the role of ORMDL3 in immune cells, as well as the contribution of other genes within the 17q12–q21 locus to T1D susceptibility.
Qiao-Dan Ou, Evan Fletcher Jones, Kennedy Currey et al.
· Biochemistry and Biophysics... · 0 citations