Glycaemic dysregulation is associated with clinically diagnosed atrial fibrillation (AF), but prior studies largely relied on intermittent or short-term monitoring of heart rhythm and glucose levels to investigate their relationship. Additionally, the relationship between glycaemic variation and AF progression ov...
A. K. Hessellund, K. Haugan, L. Xing et al.· European Heart Journal· 0 citations
Atherogenic lipoproteins are causal drivers of atherosclerotic cardiovascular disease (ASCVD). Emerging data has implicated inflammation as a key contributor to ASCVD. However, in adults without established ASCVD, it remains uncertain whether atherogenic lipid-plaque associations persists after accounting for mea...
N. Sandau, R. Hasselbalch, K. Iversen et al.· European Heart Journal· 0 citations
BACKGROUND
Long forms of cardiac troponin T (cTnT) are promising markers that may differentiate myocardial infarction from other causes of acute and chronic myocardial injury. However, to understand the clinical utility of long cTnT, knowledge of the half-life (T½) is key. By using auto-transfusion in humans we determi...
R. Hasselbalch, S. Salonen, J. Kristensen et al.· The Journal of Applied Labor...· 0 citations
Higher HbA1c levels were associated with higher AF burden and lower HbA1c levels were associated with lower AF burden and less accumulation of AF over time, and participants reaching prediabetes or diabetes accumulated longer AF durations and higher AF burden over time compared to participants with consistently normal...
A. K. Hessellund, K. Haugan, L. Y. Xing et al.· International Journal of Car...· 1 citation
Genetic predisposition to IL-1α autoantibodies is associated with reduced risk of rheumatoid arthritis and certain cancers, whereas genetic predisposition to IL-6 autoantibodies is associated with increased risk of diabetes, mirroring prior clinical observations.
J. V. von Stemann, Joseph Dowsett, R. Teglgaard et al.· Nature Communications· 0 citations
This study of silent atherosclerosis showed that the disease was detectable in early adulthood, with age- and sex-associated increases in prevalence across arterial territories and marked increases in plaque volume.
H. Bundgaard, I. García-Lunar, K. F. Kofoed et al.· New England Journal of Medic...· 0 citations
It is concluded that rare coding CNVs in a proportion of AD-associated genes and 22q11.21 duplication as a strong AD-risk-decreasing factor increase AD risk and Conversely, 22q11.21 duplication as a strong AD-risk-decreasing factor is identified.
O. Quenez, Catherine Schramm, K. Cassinari et al.· American Journal of Human Ge...· 1 citation
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