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Prenatal fluoride exposure does not appear to affect pregnancy outcomes, but preliminary evidence suggests that higher fluoride levels may affect the risk of LGA depending on timing of exposure during pregnancy.
This is the first epidemiologic study to evaluate prenatal exposure to aromatic amines in relation to infant cognition and extends concerns about aromatic amines beyond carcinogenicity to early neurodevelopment, highlighting a previously unrecognized pathway of potential harm during a critical developmental window.
The findings add to a limited evidence base on prenatal PFAS exposure and parent-reported sleep-related behaviors in early childhood and warrant confirmation in future studies that incorporate harmonized trimester-specific exposure assessment, objective sleep measures, and mixture-based analytic approaches.
Prenatal OP exposure was not associated with autism-related traits in childhood and results were consistent across sex-stratified models, secondary analyses using quantile and logistic regressions, various sensitivity analyses, and after adjustments for dietary confounders.
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