From signal to survival
This thesis shows how computational methods can aid, automate and augment the analysis of immunophenotypic characteristics in AML, enabling the translation from measurements to prognosis and outcomes (survival).
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This thesis shows how computational methods can aid, automate and augment the analysis of immunophenotypic characteristics in AML, enabling the translation from measurements to prognosis and outcomes (survival).
CD38+HLA-DR+CD8+ T cells are identified as a potential mechanistic link between Still's disease and MAS and highlight this subset as a candidate therapeutic target.
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