Intensive lipid-lowering therapy reduces cardiovascular events by lowering low-density lipoprotein cholesterol (LDL-C) and stabilizing coronary vulnerable plaques in patients with coronary artery disease (CAD). Whether similar disease-modifying effects occur in ruptured plaques of the aorta remains unknown. Non-o...
K. Kojima, K. Agune, N. Kurito et al.· European Heart Journal· 0 citations
The relationship between hypertension and aortic plaque instability detected by NOGA was evaluated and it was found that hypertension may contribute not only to plaque burden but also to plaque destabilization in the aorta.
K. Kojima, K. Agune, N. Kurito et al.· European Heart Journal· 0 citations
Background and aims Elevated lipoprotein(a) [Lp(a)] levels are recognized as a genetic risk factor for atherosclerotic cardiovascular disease. Previous coronary imaging studies have linked elevated Lp(a) levels to vulnerable coronary plaque characteristics. However, the relationship between elevated Lp(a) levels and vu...
K. Kojima, Yudai Tanaka, Katsunori Fukumoto et al.· Atherosclerosis Plus· 0 citations
Stress phase entropy derived from ECG-gated SPECT MPI was a strong long-term predictor of MCEs in Japanese patients with CAD and it is considered to be the most useful left ventricular dyssynchrony index to improve risk stratification over prediction by perfusion and LVEF assessment alone.
Katsunori Fukumoto, S. Yoda, Yudai Tanaka et al.· Internal medicine· 0 citations
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