Author

Tomohiko Taguchi

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Aug 2026

An N-terminal CDC42 T43I variant reveals the mechanism of pyrin inflammasome activation.

Heterozygous carboxyl-terminal variants in the RHO guanosine triphosphatase (GTPase) CDC42 are known to cause severe autoinflammatory syndromes. Here, we identified a heterozygous amino-terminal p.T43I (Thr43→Ile) CDC42 variant in patients with autoinflammation and uncovered a molecular link between CDC42 and the infla...

Mariko Aoki, A. Iannuzzo, P. Mertz et al. · Science immunology · 4 citations

Open access Aug 2026

Identification of a transient receptor potential channel that is regulated by phospholipid asymmetry

This work identifies TRPM8 as a phospholipid asymmetry-dependent ion channel and establishes an experimental strategy for systematically identifying membrane proteins regulated by phospholipid asymmetry, providing a foundation for future studies investigating the biological significance of this fundamental membrane pro...

R. Nakanishi, Akira Murakami, Emi Sasaki et al. · bioRxiv · 0 citations