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Noncoding genetic variants identified by the in vitro screen may play important roles in monocyte function, through the regulation of cell-surface FcγR abundance, through the regulation of cell-surface FcγR abundance.
A computational framework is developed to identify and reintegrate missing gene features, improving immune response characterization in Rhesus macaque and is broadly applicable to scRNA-seq datasets across species and experimental contexts.
The findings suggest that VILMIR modulates transcription of interferon-stimulated genes by forming an RNA-protein complex with FUBP1, an RNA-binding protein implicated in transcriptional regulation.
Two novel interferon-responsive human lncRNAs that may play important roles in the regulation of the host interferon response across different cell types are identified.
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