Neuroprotective mechanism of Ribisin A on Aβ25-35-induced PC12 cell damage model.
Evidence is provided for the neuroprotective mechanism of Ribisin A in an Aβ25-35-induced cellular injury model that can reduce oxidative damage, inhibit inflammation, restore mitochondrial function, and reduce apoptosis and may involve regulation of the TrkB-mediated ERK/CREB signaling cascade.