Polydatin Reverses Diabetes‐Induced Apoptosis of Pancreatic β‐Cells by Inhibiting the TNF/TNF‐R1/Caspase‐3 Signaling Axis
Background Pancreatic β‐apoptosis is a critical pathological event in diabetes mellitus (DM), yet effective therapeutic agents that directly protect β‐cells are lacking. Polydatin (PD), a natural stilbenoid, has shown potential in regulating glucolipid metabolism; however, its role and mechanism in protecting pancreati...