HnRNPK suppresses macrophage-driven inflammation in autoimmune myocarditis by restraining NCF1-dependent oxidized phospholipid signaling and ferroptosis-related changes
NADPH oxidase-dependent ROS production, oxidized phospholipid (OxPL) signaling, and macrophage ferroptosis are emerging drivers of inflammatory cardiac injury, yet the upstream regulatory mechanisms governing these processes remain poorly understood. Here, we identify heterogeneous nuclear ribonucleoprotein K (HnRNPK),...