Y. Shao

WTAP-induced upregulation of KDR ameliorates angiotensin II-induced apoptosis, oxidative stress and ferroptosis of human brain microvascular endothelial cells

Intracranial aneurysms (IAs) are a significant cause of morbidity and mortality, often leading to subarachnoid hemorrhage and subsequent neurological deficits. The kinase insert domain receptor (KDR) plays a crucial role in vascular cell function during IA. However, the mechanism responsible for KDR’s role in IA remain...