S100A8/S100A9 Links Diabetic Stress to Cardiac Progenitor Cell Dysfunction and Fibrotic Heart Failure: An Integrated Transcriptomic, Single‐Cell, and Functional Study
S100A8/S100A9 emerges as a central mediator linking hyperglycemia‐induced oxidative stress, metabolic inflexibility, and fibrotic reprogramming of CPCs, thereby promoting diabetic heart failure and may serve as a biomarker and therapeutic target at the interface of immunometabolism, cardiac regeneration, and cardio‐onc...