Batf2 knockdown ameliorates early-stage hyperoxia-induced BPD-like lung injury and is associated with suppression of GBP5/NF-κB signaling.
OBJECTIVE Bronchopulmonary dysplasia (BPD) is characterized by persistent inflammation and impaired alveolar development. Macrophage inflammatory dysregulation contributes to BPD progression, but its upstream regulators remain incompletely understood. This study investigated the role of Batf2 in hyperoxia-induced lung...