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This work connects lysosomal signalling with intercellular communication and suggests a broader role for RECS1 in stress-responsive secretion, which bridges lysosomal signalling and extracellular vesicles biology, revealing a new mechanism for stress-responsive cell communication.
The results reinforce the idea that targeting protein synthesis with small molecules in patients carrying C9ORF72 mutations may result in improved cognitive capacity and reinforce the idea that targeting protein synthesis with small molecules in patients carrying ALS may result in improved cognitive capacity.
Treatment of 5xFAD mice with Proteostaser-1 improved spatial learning and synaptic plasticity, and reduced the deposition of amyloid plaques in the brain, which support the therapeutic potential of the UPR as a strategy to ameliorate AD features and sustain synaptic function.
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