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Author

Senn Wakahashi

1 paper indexed here

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Open access Oct 2026

Functional redundancy between atypical CDKs reveals a vulnerability in hepatocellular carcinoma.

Findings provide a rationale for the development of CDK16-18 inhibitors-likely targeting multiple subfamily members due to their structural similarity-as a strategy against aggressive cancers, such as hepatocellular carcinoma, where effective treatment options remain limited.

Diego Martínez-Alonso, Enrique Nogueira, Luis R. López-Sainz et al. · 0 citations

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