NFAT5 regulates TGF-β-induced epithelial-mesenchymal transition in breast cancer cells through interaction with β-catenin
Findings identify NFAT5 as a contributor to TGF-β-induced EMT and stemness-associated breast cancer progression through its interaction with β-catenin, highlighting NFAT5-associated transcriptional regulation as a potential therapeutic target in aggressive breast cancer.