A conceptual framework of how the dysfunction of the highly conserved NAT10 leads to tumorigenesis, metastasis, and therapeutic resistance is provided and the major findings that reveal how NAT10 regulates cancer metabolism and immune responses are summarized.
Peng Xu, Qin Yan· Cancer Metastasis Review· 0 citations
It is shown that aberrant m ⁶ A modification is widely involved in the development of various tumors by regulating the expression of oncogenes and tumor suppressor genes, thereby influencing malignant phenotypes such as tumor proliferation, metastasis, metabolic reprogramming, immune evasion, immune evasion, and drug r...
Linxiao Feng, ,-Wu-Hong Mei, Yan Qin et al.· 0 citations
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